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WPW case 4. (With RAD and R/S >1)

Patient.

II

Characteristic:
• Ventricle is excited through the accessory pathway sooner than normally excited through the normal conduction pathway, creating: (1) short PR interval (<120 ms), (2) Wide QRS, begin with Delta wave, (3) Abnormal (secondary) repolarization.
Other findings:
• Ventricular preexcitation may produce pseudo (infarction) Q wave (4), case 2.
• Ventricular preexcitation may produce abnormal QRS voltage, abnormal axis on frontal plane (LAD in case 2 and RAD in case 4), and abnormal QRS direction in precordial leads. (5) Predominant R wave in V1 (R/S ratio >1 in V1) in case 1 and case 4.
• Repolarization change may mask other cause of abnormal repolarization.
• May display varying degree of preexcitation, observed in the same patient. (Case 2 and Case 3)

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WPW case 4. (With RAD and R/S >1)
Patient.
II	Characteristic: • Ventricle is excited through the accessory pathway sooner than normally excited through the normal conduction pathway, creating: (1) short PR interval (<120 ms), (2) Wide QRS, begin with Delta wave, (3) Abnormal (secondary) repolarization. Other findings: • Ventricular preexcitation may produce pseudo (infarction) Q wave (4), case 2. • Ventricular preexcitation may produce abnormal QRS voltage, abnormal axis on frontal plane (LAD in case 2 and RAD in case 4), and abnormal QRS direction in precordial leads. (5) Predominant R wave in V1 (R/S ratio >1 in V1) in case 1 and case 4. • Repolarization change may mask other cause of abnormal repolarization. • May display varying degree of preexcitation, observed in the same patient. (Case 2 and Case 3)